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Results: 3

1.
Figure 1

Figure 1. From: High-Intensity Physical Activity Modulates Diet Effects on Cerebrospinal ?-Amyloid Levels in Normal Aging and Mild Cognitive Impairment.

Model of hypothetical trajectory of brain and CSF Aβ42 with increasing AD symptoms and pathology [27].

Laura D. Baker, et al. J Alzheimers Dis. ;28(1):137-146.
2.
Figure 3

Figure 3. From: High-Intensity Physical Activity Modulates Diet Effects on Cerebrospinal ?-Amyloid Levels in Normal Aging and Mild Cognitive Impairment.

Scatter plots and least squares regression lines depicting baseline associations between, A) min/wk hi–PA and CSF concentration of total tau protein (pg/mL, log transformed, n=18), r=−0.54, p=0.020, B) min/wk hi–PA and CSF concentration of IL-8 (pg/mL, n=10), r=−0.70, p=0.025 and C) CSF concentrations of total tau protein (pg/mL, log transformed) and IL-8 (pg/mL), r=0.87, p=0.001.

Laura D. Baker, et al. J Alzheimers Dis. ;28(1):137-146.
3.
Figure 2

Figure 2. From: High-Intensity Physical Activity Modulates Diet Effects on Cerebrospinal ?-Amyloid Levels in Normal Aging and Mild Cognitive Impairment.

Means and SEM representing change from baseline to week 4 for CSF levels of Aβ42, expressed as residual scores, for normal adults and adults with MCI by high-intensity physical activity (hi–PA) groups created using a median split on min/wk. In all analyses, min/wk hi–PA was entered as a continuous variable. Categorical hi–PA groups depicted in this figure were created solely for the purpose of illustration. A significant 3-way interaction involving diet, diagnosis, and hi–PA on CSF Aβ42 (ANOVA, p=0.039) and the results of follow-up analyses indicated that the HIGH diet-induced increase in CSF Aβ42 was attenuated for normal adults completing more min/wk hi–PA (r=−0.61, p=0.11), while in MCI, the LOW diet effect on CSF Aβ42 was potentiated by increasing amounts of hi–PA (r=0.64, p=0.034). There were no baseline differences between groups.

Laura D. Baker, et al. J Alzheimers Dis. ;28(1):137-146.

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