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1.
Fig. 5

Fig. 5. Changes in Levels of Protein Carbonyls. From: Sleep Deprivation Under Sustained Hypoxia Protects Against Oxidative Stress.

Changes in levels of protein carbonyls in the neocortex, hippocampus, brainstem and cerebellum of rats exposed to sleep deprivation under sustained hypoxia (SDSH) versus control animals left undisturbed under either sustained hypoxia (UCSH) or normoxia (UCN). Data are expressed as mean ± S.E.M.

Lalini Ramanathan, et al. Free Radic Biol Med. ;51(10):1842-1848.
2.
Fig. 6

Fig. 6. Changes in Hexokinase(HK) Activity. From: Sleep Deprivation Under Sustained Hypoxia Protects Against Oxidative Stress.

Changes in hexokinase (HK) activity in the neocortex, hippocampus, brainstem and cerebellum of rats exposed to sleep deprivation under sustained hypoxia (SDSH) versus control animals left undisturbed under either sustained hypoxia (UCSH) or normoxia (UCN). Data are expressed as mean ± S.E.M. a= SDSH vs UCSH, p<0.05.

Lalini Ramanathan, et al. Free Radic Biol Med. ;51(10):1842-1848.
3.
Fig. 2

Fig. 2. Changes in Total Glutathione (GSHt) Levels. From: Sleep Deprivation Under Sustained Hypoxia Protects Against Oxidative Stress.

Changes in total glutathione (GSHt) levels in the neocortex, hippocampus, brainstem and cerebellum of rats exposed to sleep deprivation under sustained hypoxia (SDSH) versus control animals left undisturbed under either sustained hypoxia (UCSH) or normoxia (UCN). Data are expressed as mean ± S.E.M. a= SDSH vs UCSH, p<0.05; b= SDSH vs UCN, p<0.05

Lalini Ramanathan, et al. Free Radic Biol Med. ;51(10):1842-1848.
4.
Fig. 1

Fig. 1. Changes in Nitric Oxide Levels. From: Sleep Deprivation Under Sustained Hypoxia Protects Against Oxidative Stress.

Changes in nitric oxide (NO) levels in the neocortex, hippocampus, brainstem and cerebellum of rats exposed to sleep deprivation under sustained hypoxia (SDSH) versus control animals left undisturbed under either sustained hypoxia (UCSH) or normoxia (UCN). Data are expressed as mean ± S.E.M. a= SDSH vs UCSH, p<0.05; b= SDSH vs UCN, p<0.05.

Lalini Ramanathan, et al. Free Radic Biol Med. ;51(10):1842-1848.
5.
Fig. 3

Fig. 3. Changes in Superoxide Dismutase (SOD) Activity. From: Sleep Deprivation Under Sustained Hypoxia Protects Against Oxidative Stress.

Changes in superoxide dismutase (SOD) activity in the neocortex, hippocampus, brainstem and cerebellum of rats exposed to sleep deprivation under sustained hypoxia (SDSH) versus control animals left undisturbed under either sustained hypoxia (UCSH) or normoxia (UCN). Data are expressed as mean ± S.E.M.

Lalini Ramanathan, et al. Free Radic Biol Med. ;51(10):1842-1848.
6.
Fig. 4

Fig. 4. Changes in Levels of TBARS. From: Sleep Deprivation Under Sustained Hypoxia Protects Against Oxidative Stress.

Changes in levels of thiobarbituric acid reactive substances (TBARS) in the neocortex, hippocampus, brainstem and cerebellum of rats exposed to sleep deprivation under sustained hypoxia (SDSH) versus control animals left undisturbed under either sustained hypoxia (UCSH) or normoxia (UCN). Data are expressed as mean ± S.E.M. a= SDSH vs UCSH, p<0.05.

Lalini Ramanathan, et al. Free Radic Biol Med. ;51(10):1842-1848.

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