Results: 4

1.
Figure 2

Figure 2. From: The Role of DNA Repair Capacity in Melanoma Skin Cancer Risk in a Population Chronically Exposed to High Levels of Sunlight.

DNA repair capacity (DRC) of controls in terms of their type (in situ and malignant melanoma). Values are expressed as the mean ± standard error of the mean and adjusted by age.

Jaime L. Matta, et al. Ochsner J. 2010 Summer;10(2):75-82.
2.
Figure 3

Figure 3. From: The Role of DNA Repair Capacity in Melanoma Skin Cancer Risk in a Population Chronically Exposed to High Levels of Sunlight.

DNA repair capacity (DRC) of controls versus cases as a function of tumor depth, based on the Breslow index, with corresponding P value obtained through a Student's t test.

Jaime L. Matta, et al. Ochsner J. 2010 Summer;10(2):75-82.
3.
Figure 4

Figure 4. From: The Role of DNA Repair Capacity in Melanoma Skin Cancer Risk in a Population Chronically Exposed to High Levels of Sunlight.

The association between DNA repair capacity (DRC) and the anatomical location of both in situ and malignant melanoma tumors in terms of sun exposure was analyzed. Patients with melanoma skin cancer in sun-exposed areas had a 38% reduction in DRC when compared to controls with borderline significance, P  =  0.055.

Jaime L. Matta, et al. Ochsner J. 2010 Summer;10(2):75-82.
4.
Figure 1

Figure 1. From: The Role of DNA Repair Capacity in Melanoma Skin Cancer Risk in a Population Chronically Exposed to High Levels of Sunlight.

Age-adjusted percentage DNA repair capacity (DRC) of persons in Puerto Rico without skin cancer (n  =  124) and with melanoma skin cancer (n  =  62). DRC was measured in lymphocytes by using the host-cell reactivation assay with a luciferase reporter gene according to Matta et al.18 Values are expressed as the mean ± standard error of the mean. Patients with melanoma had a 3% reduction in DRC, as compared to controls, that was not statistically significant (P  =  .697, Student's t test).

Jaime L. Matta, et al. Ochsner J. 2010 Summer;10(2):75-82.

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