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3.
Fig. 5

Fig. 5. From: Effects of epigallocatechin gallate, L-ascorbic acid, ?-tocopherol and dihydrolipoic acid on the formation of deoxyguanosine adducts derived from lipid peroxidation.

Effects of various concentrations of EGCG (A), ascorbic acid (B), α-tocopherol (C) and DHLA (D) on the formation of 8-oxodG in the reaction of DHA with dG in the presence of FeSO4. The values are mean ± standard deviation from 3-4 experiments. *significantly different from the control

Raghu G. Nath, et al. Nutr Cancer. 2010;62(5):622-629.
4.
Fig. 4

Fig. 4. From: Effects of epigallocatechin gallate, L-ascorbic acid, ?-tocopherol and dihydrolipoic acid on the formation of deoxyguanosine adducts derived from lipid peroxidation.

Effects of various concentrations of EGCG (A), ascorbic acid (B), α-tocopherol (C) and DHLA (D) on the formation of γ-OH-Acr-dG in the reaction of DHA with dG in the presence of FeSO4. The values are mean ± standard deviation from 3-4 experiments. *significantly different from the control; n.d., not detectable

Raghu G. Nath, et al. Nutr Cancer. 2010;62(5):622-629.
5.
Fig. 6

Fig. 6. From: Effects of epigallocatechin gallate, L-ascorbic acid, ?-tocopherol and dihydrolipoic acid on the formation of deoxyguanosine adducts derived from lipid peroxidation.

Effects of various concentrations of EGCG (A), ascorbic acid (B), α-tocopherol (C) and DHLA (D) on the formation of Acr-dG adducts (total of α-OH-Acr-dG and γ-OH-Acr-dG) in the reaction of Acr with dG. The values are mean ± standard deviation from 3-4 experiments. *significantly different from the control; n.d., not detected

Raghu G. Nath, et al. Nutr Cancer. 2010;62(5):622-629.
6.
Fig. 3

Fig. 3. From: Effects of epigallocatechin gallate, L-ascorbic acid, ?-tocopherol and dihydrolipoic acid on the formation of deoxyguanosine adducts derived from lipid peroxidation.

Typical HPLC chromatograms showing the analysis of the reaction mixture of DHA (3 mM) and dG (15 mM) in the presence of FeSO4 (Panel A) using System 1. The peaks at 26.7 and 33.3 min represent 8-oxodG and γ-OH-Acr-dG, respectively. α-OH-Acr-dG was not detected in this reaction. Panel B shows the analysis of the reaction mixture of Acr (0.5 mM) and dG (0.5 mM) using HPLC System 3. The peaks at 45.0 and 48.2 and 49.5 min represent α-OH-Acr-dG and γ-OH-Acr-dG, respectively.

Raghu G. Nath, et al. Nutr Cancer. 2010;62(5):622-629.

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