Results: 2

1.
Figure 1

Figure 1. From: Endotoxemia-induced inflammation and the effect on the human brain.

LPS-induced changes in cytokine plasma concentrations, cortisol and brain specific proteins. Time -0- reflects baseline concentrations. Administration of lipopolysaccharide (LPS) resulted in a marked increase in TNF-α, IL-6, IL-10, IL-1Ra and cortisol concentrations. All changes in cytokine and the cortisol concentrations were significant (P < 0.001). Concentrations of neuron specific enolase (NSE) decreased after administration of LPS (P < 0.001) and S100-β showed a significant biphasic change (P = 0.038). All data are expressed as mean ± standard error of the mean (n = 15). GFAP, glial fibrillary acidic protein; S100β, S100 Calcium Binding Protein B. * P < 0.05. ** P < 0.001.

Mark van den Boogaard, et al. Crit Care. 2010;14(3):R81-R81.
2.
Figure 2

Figure 2. From: Endotoxemia-induced inflammation and the effect on the human brain.

Increase of the EEG occipital peak frequencies, relative alpha band power and absolute alpha and beta band power two to three hours after LPS infusion. Data of peak frequency are absolute numbers, data of absolute and relative band power are expressed as percentage changes. Time -0- reflects baseline measurements. (standard error of the means were omitted for reasons of clarity). * P < 0.05. ** P < 0.001. (a) Peak frequency in occipital region. Friedman analysis of variance revealed changes in P4O2 and P3O1 (both P < 0.001). (b) Percentage change compared to baseline in absolute band power (ABP) of alpha activity in occipital and central region. Friedman analysis of variance revealed changes for alpha activity in P4O, P3O1 and F4C4, F3C3 all P < 0.001. (c) Percentage change compared with baseline in absolute band power (ABP) and relative band power (RBP) of beta activity in occipital region. Friedman analysis of variance revealed changes of RBP for beta activity in P4O2 (P = 0.017), P3O1 (P = 0.575) and ABP for beta activity in P4O and P3O1 (both P < 0.001).

Mark van den Boogaard, et al. Crit Care. 2010;14(3):R81-R81.

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