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Results: 5

1.
FIGURE 2.

FIGURE 2. From: Acute Coronary Syndromes: Diagnosis and Management, Part I.

Algorithm for evaluation and management of patients with suspected acute coronary syndrome (ACS). ACC = American College of Cardiology; AHA = American Heart Association; ECG = electrocardiography; LV = left ventricular.
From J Am Coll Cardiol,42 with permission from Elsevier.

Amit Kumar, et al. Mayo Clin Proc. 2009 October;84(10):917-938.
2.
FIGURE 5.

FIGURE 5. From: Acute Coronary Syndromes: Diagnosis and Management, Part I.

Long-term antithrombotic therapy at hospital discharge after unstable angina (UA)/non—ST-segment elevation myocardial infarction (NSTEMI). LOE = level of evidence.
a For patients allergic to aspirin, use clopidogrel alone indefinitely, or try aspirin desensitization.
b For patients allergic to clopidogrel, use ticlopidine, 250 mg by mouth twice daily.
c Continue aspirin indefinitely and warfarin longer term as indicated for such specific conditions as atrial fibrillation; left ventricular thrombus; and cerebral, venous, or pulmonary emboli.
d When warfarin is added to aspirin plus clopidogrel, an international normalized ratio of 2.0 to 2.5 is recommended.
From J Am Coll Cardiol,42 with permission from Elsevier.

Amit Kumar, et al. Mayo Clin Proc. 2009 October;84(10):917-938.
3.
FIGURE 4.

FIGURE 4. From: Acute Coronary Syndromes: Diagnosis and Management, Part I.

Algorithm for patients with UA/NSTEMI managed by an initial conservative strategy. When multiple drugs are listed, they are in alphabetical order and not in order of preference. EF = ejection fraction; GP = glycoprotein; LOE = level of evidence; LVEF = left ventricular ejection fraction; NSTEMI = non—ST-segment elevation myocardial infarction; UA = unstable angina; UFH = unfractionated heparin.
a For full dosing information, see Table 13 in reference 42.
b For example, recurrent symptoms/ischemia, heart failure, or serious arrhythmia.
c For more details on management of patients with UA/NSTEMI after diagnostic angiography, see Figure 9 of reference 42.
d See recommendations in section 3.2.3 of reference 42.
From J Am Coll Cardiol,42 with permission from Elsevier.

Amit Kumar, et al. Mayo Clin Proc. 2009 October;84(10):917-938.
4.
FIGURE 3.

FIGURE 3. From: Acute Coronary Syndromes: Diagnosis and Management, Part I.

Algorithm for patients with UA/NSTEMI managed by an initial invasive strategy. When multiple drugs are listed, they are in alphabetical order and not in order of preference. GP = glycoprotein; IV = intravenous; LOE = level of evidence; NSTEMI = non—ST-segment elevation myocardial infarction; UA = unstable angina; UFH = unfractionated heparin.
a For full dosing information, see Table 13 in reference 42.
b Evidence exists that GP IIb/IIIa inhibitors may not be necessary if the patient received a preloading dose of at least 300 mg of clopidogrel at least 6 h earlier (class I, LOE: B for clopidogrel administration) and bivalirudin is selected as the anticoagulant (class IIa, LOE: B).
cFor more details on management of patients with UA/NSTEMI after diagnostic angiography, see Figure 9 of reference 42.
From J Am Coll Cardiol,42 with permission from Elsevier.

Amit Kumar, et al. Mayo Clin Proc. 2009 October;84(10):917-938.
5.
FIGURE 1.

FIGURE 1. From: Acute Coronary Syndromes: Diagnosis and Management, Part I.

Timing of release of various biomarkers after acute myocardial infarction (AMI). The biomarkers are plotted showing the multiples of the cutoff for AMI over time. The dashed horizontal line shows the upper limit of normal (ULN, defined as the 99th percentile from a normal reference population without myocardial necrosis; the coefficient of variation [CV] of the assay should be 10% or less). The earliest rising biomarkers are myoglobin and creatine kinase (CK) isoforms (leftmost curve). The muscle and brain fraction of CK (CK-MB, dashed curve) rises to a peak of 2 to 5 times the ULN and typically returns to the normal range within 2 to 3 d after AMI. The cardiac-specific troponins show small elevations above the ULN in small infarctions (eg, as is often the case with non—ST-segment elevation MI) but rise to 20 to 50 times the ULN in the setting of large infarctions (eg, as is typically the case in ST-segment elevation MI). The troponin levels may stay elevated above the ULN for 7 d or more after AMI.
Adapted from Mayo Clinic Cardiology: Concise Textbook, 3rd ed.58

Amit Kumar, et al. Mayo Clin Proc. 2009 October;84(10):917-938.

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