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1.
Figure 2

Figure 2. From: Use of Quantitative EEG in Infants with Port-Wine Birthmark to Assess for Sturge-Weber Brain Involvement.

Flow diagram of subject recruitment and categorization

Joshua B. Ewen, et al. Clin Neurophysiol. ;120(8):1433-1440.
2.
Figure 4

Figure 4. From: Use of Quantitative EEG in Infants with Port-Wine Birthmark to Assess for Sturge-Weber Brain Involvement.

This diagram shows the topographical distribution of power in each frequency band for subject V-5. Power is reduced on the left side. Note the different power scale for delta/theta vs. alpha/beta.

Joshua B. Ewen, et al. Clin Neurophysiol. ;120(8):1433-1440.
3.
Figure 1

Figure 1. From: Use of Quantitative EEG in Infants with Port-Wine Birthmark to Assess for Sturge-Weber Brain Involvement.

This EEG in a patient is clearly asymmetrical, with decreased amplitude over the left (first and third groupings of 4 channels each).

Joshua B. Ewen, et al. Clin Neurophysiol. ;120(8):1433-1440.
4.
Figure 3

Figure 3. From: Use of Quantitative EEG in Infants with Port-Wine Birthmark to Assess for Sturge-Weber Brain Involvement.

Three month-old boy with bilateral intracranial involvement of SWS, right greater than left. Axial T2-weighted images (top panel) show moderate to marked right and mild left cerebral atrophy. Post-Gadolinium T1-weighted images (bottom panel) demonstrate leptomeningeal angiomatosis in all lobes of the right hemisphere as well as left temporal and occipital lobes (see arrows, imaging score R:15; L:7).

Joshua B. Ewen, et al. Clin Neurophysiol. ;120(8):1433-1440.

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