Results: 4

1.
Figure 4

Figure 4. From: Post-cardiac arrest temperature manipulation alters early EEG bursting in rats.

Early burst frequency correlated strongly with 72-hour NDS in normothermic rats (p<0.01)

Xiaofeng Jia, et al. Resuscitation. ;78(3):367-373.
2.
Figure 2

Figure 2. Raw EEG data of representative rats in the 3 temperature groups. From: Post-cardiac arrest temperature manipulation alters early EEG bursting in rats.

Burst-suppression after CA recovered slower in normothermic rats than hypothermic and hypothermic rats.

Xiaofeng Jia, et al. Resuscitation. ;78(3):367-373.
3.
Figure 3

Figure 3. Early burst frequency post-resuscitation in the 3 temperature groups. From: Post-cardiac arrest temperature manipulation alters early EEG bursting in rats.

(*p<0.05, **p<0.01, ***p<0.001) The hypothermia group had a significantly higher burst frequency than both the normothermia and hyperthermia groups between 30 and 60 minutes post-resuscitation.

Xiaofeng Jia, et al. Resuscitation. ;78(3):367-373.
4.
Figure 1

Figure 1. Temperature recording of 7-minute asphyxial CA rats. From: Post-cardiac arrest temperature manipulation alters early EEG bursting in rats.

The different cohorts received hyperthermia (upper), normothermia (middle), and hypothermia (bottom). The plots show temperatures during four different phases of the experiment. A: baseline and asphyxial CA period, B: temperature manipulation induction period, C: temperature manipulation maintenance period, and D: temperature manipulation recovery period.

Xiaofeng Jia, et al. Resuscitation. ;78(3):367-373.

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