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Results: 3

1.
FIG. 2.

FIG. 2. From: Whole-Genome Analyses of Speciation Events in Pathogenic Brucellae .

Venn diagram displaying the distribution of pseudogenes among the Brucella genomes. The total number of pseudogenes in each section is shown in blue; distribution by chromosome is indicated (ChrI/ChrII). The total number of pseudogenes within each species genome is shown outside the circles and under the species names. Note that these numbers do not reflect genes that are absent from the genomes.

Patrick S. G. Chain, et al. Infect Immun. 2005 December;73(12):8353-8361.
2.
FIG. 1.

FIG. 1. From: Whole-Genome Analyses of Speciation Events in Pathogenic Brucellae .

Whole-genome comparison of B. abortus 2308, B. melitensis 16M, and B. suis 1330. A linear representation of both chromosomes displays the approximate locations and sizes (in kilobases, below features) of major insertions, deletions, and repetitive elements (including IS711 and rRNA sequences, as well as repeated phage and IS3 family elements). Also indicated are the approximate locations and sizes (in kilobases, above features) of inversions found within the chromosomes of the three Brucella spp. The hashed boxes indicate regions that were found to be missing from B. abortus strain 2308 but were present in strain 9-941s. Note that the large inversion in ChrII of B. abortus has been flipped in this diagram for better visual alignment (of the features within this inversion) with the other genomes. Refer to the inset for definitions of genome features.

Patrick S. G. Chain, et al. Infect Immun. 2005 December;73(12):8353-8361.
3.
FIG. 3.

FIG. 3. From: Whole-Genome Analyses of Speciation Events in Pathogenic Brucellae .

Distribution of pseudogenes by functional category. Functional classifications are as follows: J, translation ribosomal structure and biogenesis; K, transcription; L, DNA replication, recombination, and repair; D, cell division; V, defense mechanisms; T, signal transduction; M, cell envelope, biogenesis, and outer membrane; N, cell motility and secretion; U, intracellular traffic, secretion, and vesicular transport; O, posttranslational modification, protein turnover, and chaperons; C, energy production and conversion; G, carbohydrate metabolism; E, amino acid metabolism; F, nucleotide metabolism; H, coenzyme metabolism; I, lipid metabolism; Q, secondary metabolite biosynthesis and catabolism; R, general function prediction only; S, function unknown; X, no cluster of orthologous groups (COG); TR, transport.

Patrick S. G. Chain, et al. Infect Immun. 2005 December;73(12):8353-8361.

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