Display Settings:

Items per page

Results: 8

1.

Figure. From: Effects of Interleukin-1? Administration on Intestinal Ischemia and Reperfusion Injury, Mucosal Permeability, and Bacterial Translocation in Burn and Sepsis.

Figure 4. Alterations in mesenteric vascular resistance (MVR) after burn (0 hour) and endotoxin (18 hours). IL-1α had a positive impact on MVR. P < .05 *vs. baseline, #vs. sham, ¥vs. IL-1α.

Tamer Tadros, et al. Ann Surg. 2003 January;237(1):101-109.
2.

Figure. From: Effects of Interleukin-1? Administration on Intestinal Ischemia and Reperfusion Injury, Mucosal Permeability, and Bacterial Translocation in Burn and Sepsis.

Figure 5. Administration of IL-1α slightly affected systemic oxygen delivery (DO2) and oxygen consumption (VO2) after burn (0 hour) and endotoxin (18 hours). P < .05 *vs. baseline, #vs. sham, ¥vs. IL-1α.

Tamer Tadros, et al. Ann Surg. 2003 January;237(1):101-109.
3.

Figure. From: Effects of Interleukin-1? Administration on Intestinal Ischemia and Reperfusion Injury, Mucosal Permeability, and Bacterial Translocation in Burn and Sepsis.

Figure 6. Burn (0 hour) and endotoxin (18 hours) significantly reduced mesenteric oxygen supply (mDO2) and oxygen consumption (mVO2). These detrimental effects were attenuated by IL-1α treatment. P < .05 *vs. baseline, #vs. sham, ¥vs. IL-1α.

Tamer Tadros, et al. Ann Surg. 2003 January;237(1):101-109.
4.

Figure. From: Effects of Interleukin-1? Administration on Intestinal Ischemia and Reperfusion Injury, Mucosal Permeability, and Bacterial Translocation in Burn and Sepsis.

Figure 7. Intestinal permeability, as assessed by the lactulose/mannitol (L/M) excretion ratio, was significantly increased after burn (0 hour) and endotoxin (18 hours). IL-1α administration significantly reduced intestinal permeability. P < .05 *vs. baseline, #vs. sham, ¥vs. IL-1α.

Tamer Tadros, et al. Ann Surg. 2003 January;237(1):101-109.
5.

Figure. From: Effects of Interleukin-1? Administration on Intestinal Ischemia and Reperfusion Injury, Mucosal Permeability, and Bacterial Translocation in Burn and Sepsis.

Figure 3. Superior mesenteric artery (SMA) blood flow was significantly reduced and showed a pattern of ischemia and reperfusion after burn (0 hour) and endotoxin (18 hours). IL-1α administration significantly improved SMA blood flow. P < .05 *vs. baseline, #vs. sham, ¥vs. IL-1α.

Tamer Tadros, et al. Ann Surg. 2003 January;237(1):101-109.
6.

Figure. From: Effects of Interleukin-1? Administration on Intestinal Ischemia and Reperfusion Injury, Mucosal Permeability, and Bacterial Translocation in Burn and Sepsis.

Figure 1. Mean arterial pressure (MAP) and central venous pressure (CVP) after burn (0 hour) and endotoxin (18 hours). IL-1α treatment had a marginal effect. P < .05 *vs. baseline, #vs. sham, ¥vs. IL-1α.

Tamer Tadros, et al. Ann Surg. 2003 January;237(1):101-109.
7.

Figure. From: Effects of Interleukin-1? Administration on Intestinal Ischemia and Reperfusion Injury, Mucosal Permeability, and Bacterial Translocation in Burn and Sepsis.

Figure 2. IL-1α ameliorated the changes in cardiac output (CO) and systemic vascular resistance index (SVRI) postburn (0 hour) and early after endotoxin (18 hours). P < .05 *vs. baseline, #vs. sham, ¥vs. IL-1α.

Tamer Tadros, et al. Ann Surg. 2003 January;237(1):101-109.
8.

Figure. From: Effects of Interleukin-1? Administration on Intestinal Ischemia and Reperfusion Injury, Mucosal Permeability, and Bacterial Translocation in Burn and Sepsis.

Figure 8. Incidence of bacterial translocation to remote organs was significantly increased in the burn and endotoxin group. IL-1α treatment yielded a significant reduction in the rates of positive tissue cultures with enteric bacteria. Data are presented as percentages of harvested tissue samples. P < .05 #vs. sham, ¥vs. IL-1α.

Tamer Tadros, et al. Ann Surg. 2003 January;237(1):101-109.

Display Settings:

Items per page

Supplemental Content

Recent activity

Your browsing activity is empty.

Activity recording is turned off.

Turn recording back on

See more...
Write to the Help Desk