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Items: 2

1.

Expression data from pulmonary Type 2 Myeloid (T2M) cells, Eosinophils, Neutrophils, and Macrophages, from IL-25 treated 4get mice

(Submitter supplied) Many symptoms associated with allergic asthma result from the sequelae of type 2 inflammation. Interleukin (IL)-25 promotes type 2 inflammatory responses, and T2M cells represent an IL-4 and IL-13 producing granulocytic IL-25 responsive population. We used microarrays to characterize the gene expression profile of T2M cells, and compared T2M cells to other inflammatory subsets (eosinophils, neutrophils, and macrophages) in the lungs of mice with IL-25-induced pulmonary inflammation.
Organism:
Mus musculus
Type:
Expression profiling by array
Dataset:
GDS4422
Platform:
GPL1261
9 Samples
Download data:
GEO (CEL)
Series
Accession:
GSE36392
ID:
200036392
2.
Full record GDS4422

Interleukin-25-induced pulmonary inflammation model: Type 2 myeloid cells and other inflammatory subsets

Analysis of inflammatory cell types from lungs of 4get females that received daily intra-tracheal injections of IL-25 for 4 days to induce antigen-independent type 2 inflammation. Results provide insight into role of IL-25 and its receptor in innate and adaptive pulmonary type 2 immune responses.
Organism:
Mus musculus
Type:
Expression profiling by array, transformed count, 4 cell type sets
Platform:
GPL1261
Series:
GSE36392
9 Samples
Download data:
GEO (CEL)

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