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Genome-wide uH2A localization analysis highlights Bmi1-dependent deposition of the mark at repressed genes.

ESS000014 - Study

Summary

ChIP-seq analysis of ubiquitylation of histone H2A in wild-type mouse embryonic fibroblasts and mouse embryonic fibroblasts that are mutant for bmi1, a component of the PRC1 complex. Microarray analysis to examine gene expression is also performed.

Keywords: histone modification, expression, microarray, ChIP-seq, polycomb repressive complexes, ChIP-Seq, ubiquitin

Samples

Mouse fibroblast, embryo

ESM000120 - Similar

Mouse fibroblast, embryo

ESM000121 - Similar

Total: 2 samples - View details

Experiments

Total: 2 experiments - View detailsDownload tracksView on genome

Data submission

Gene expression and UH2A ChIP-Seq binding analysis in Bmi1 knock-out and wild type MEFs

This SuperSeries is composed of the following subset Series: GSE15715: Gene expression changes in Bmi1 knock-out MEFs as compared to wild-type. GSE15896: Genome wide uH2A localization analysis highlights Bmi1-dependent deposition of the mark at repressed genes.

Laboratory of Molecular Immunology/National Heart Lung and Blood Institute/National Institutes of Health. Public on: May 20 2009

GSE15909 - GEOSRA

Reference

Genome-wide uH2A localization analysis highlights Bmi1-dependent deposition of the mark at repressed genes.

Kallin EM, et al. PLoS Genet. 2009 Jun;5(6):e1000506. - Similar

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